Lipolyze E2 can be best categorized as a caloric wasting compound. Whereas most fat burning formulations on today's market depend on stimulants such as caffeine, and synephrine, Species Nutrition rejects the premise that the only effective way to burn fat is to "stimulate" the body into submission. While stimulants do increase metabolic rate, they also increase the production of catabolic (muscle wasting) hormones such as cortisol. As cortisol levels rise in the body, lean muscle tissue is broken down, immune system function is suppressed and dangerous abdominal belly fat deposition is increased.
Remember, the quantity of lean muscle in the body is highly correlated with the basal metabolic rate (BMR). Less muscle means a lower BMR. If stimulant-based fat burners are used to help shed unwanted body fat, there's a very good chance that, once they're discontinued, metabolic rate will be significantly lower and the predisposition to gain unwanted body fat will be significantly higher. If this scenario wasn't bad enough, consider this, stimulants raise blood pressure, increase heart rate, and increase anxiety levels. Therefore, individuals with a history of heart disease, high blood pressure, or brain chemistry abnormalities should certainly steer clear of these compounds. Lipolyze combines five unique herbal compounds for an extremely potent, non-stimulant based fat burning effect.
OEA is is an endogenous peroxisome proliferator-activated receptor alpha (PPAR-á) agonist synthesized in the intestines. PPAR-á is highly expressed in muscle, the liver, kidneys, and heart and is involved in the regulation of lipid metabolism, specifically the transcription of the genes involved in the beta-oxidation (burning) of fatty acids and lipogenesis (storage of fatty acids).
OEA has also been shown to have anorexic properties, meaning it decreases food intake and sends hunger-curbing messages to the brain. If all that weren't enough, OEA can also lower lower blood cholesterol and triglyceride levels.
T2 (3,5 di-iodo-thyronine)*
T2 is a metabolite of the thyroid hormones T4 (thyroxine) & T3 (triiodothyronine). It has specific direct actions which do not involve thyroid hormone receptors, unlike T4 and T3.
(1) It raises BMR (basal metabolic rate) which in turn increases energy expenditure in the body.
(2) It's a metabolic uncoupling agent that "uncouples" or disconnects the ATP-producing machinery in the cell and "releases" all the potential energy that was supposed to be used to produce ATP. Due to the energy and ATP loss (which is dissipated as heat), the muscle cells must dig into stored body fat stores and oxidize additional fats to ensure that optimal energy levels are maintained.
The following analogy should help explain how T2 exerts its energy-wasting effect in the human body. Imagine someone putting a hole in the gas tank of your automobile. The car would still perform at maximal capacity however, it would require a significant amount of additional fuel to travel the same distance it had prior to the punctured tank. Likewise, T2 makes the mitochondrial membranes more permeable (creates openings) which cause the loss of valuable "fuel" (ATP) from the "tank" (muscle cell). To combat this energy loss, additional fatty acids must be oxidized to ensure optimal performance levels.
Even more exciting is that fact that T2 "wastes" calories without elevating blood pressure, without raising heart rate, and without causing central nervous system over-stimulation.
(3)T2 stimulates the conversion of T4 (inactive thyroid hormone) into the more active T3 form.
(4)T2 stimulates the release of growth hormone (GH) which is a powerful fat burning and tissue regenerating compound.
Inside the cell, L-carnitine transports long chain fats into the mitochondria (energy-producing chamber) where they are then oxidized into ATP. In theory, if we can increase the amount of L-carnitine within the muscle, we can theoretically increase the quantity of fatty acids that get oxidized. Until recently the problem has been that regular L-carnitine and even the more exotic acetyl-L-carnitine lacked the ability to penetrate skeletal muscle tissue (which is where most fat oxidation occurs).
On the other hand, Propionyl-L-carnitine, a relatively new version of L-carnitine, has important effects on skeletal and cardiac muscle. Back in 1990, a human study demonstrated that propionyl-L-carnitine could combat the destructive effects of low oxygen status and muscle fatigue in heart and skeletal muscle. More relevant to our discussion is the fact that propionyl-L-carnitine has a tremendous ability to penetrate skeletal muscle and thus facilitate the transport of long chain fatty acids into the mitochondria.
The thyroid gland releases a hormone known commonly as thyroxin (T4 for short). T4 is an inactive form of thyroid hormone that must ultimately be converted into triiodothyronine (T3), the active thyroid hormone. Whenever any sort of diet is initiated, the body's natural inclination is to slow down the conversion of T4 (inactive) to T3 (active). Ultimately, when this happens, metabolic rate slows and fat losses crawl to a halt.
In India, the Guggul is a tree which exudes a resinous sap out of incisions that are made in its bark. This resin has been used for centuries as part of Indias traditional medicine called Ayurveda. This resinous sap can be processed, purified, and then standardized for its active components Guggulsterones Z and Guggulsterones E. These two fractionations have the unique ability to support thyroid function by increasing the conversion of inactive T4 into active T3 and, in doing so, restore normal thyroid function to the dieting athlete.
Green Tea Extract*
Green Tea Extract (Camellia sinesis leaf) is a powerful antioxidant similar to vitamin C, vitamin E, and beta-carotene. However, researchers have suggested that the active ingredient called epigallocatechin gallate (EGCG) may, amazingly, have 200 times the antioxidant properties of vitamin E. A study from the American Journal of Clinical Nutrition revealed that green tea extract caused a significant increase in energy expenditure and fatty acid oxidation. The researchers noted a 4% increase in 24-hour energy expenditure which actually translates to a 35-43% increase in daytime thermogenesis. What's even more significant to cardiac and thyroid patients is the fact that none of the research subjects reported any of the side effects rapid heart rate, elevated blood pressure, or increased anxiety that are commonly seen with the multitude of stimulant-based fat burners.
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